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This pathway involves the metabolism of methylglyoxal (MGO) into lactoylglutathione (LGSH) by glyoxalase I (GLO1) with glutathione (GSH), followed by hydrolysis via glyoxalase II (GLO2) to yield D-lactate

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In the present work, GHK-Cu did not change NO secretion (Supplementary Figure S1B)

Stress Management: High stress levels accelerate oxidative damage

Crystal structures of two practically distinct SusCD complexes purified from B

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