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Safety and efficacy of plerixafor dose escalation for the mobilization of CD34(+) hematopoietic progenitor cells in patients with sickle cell disease: interim results

Thus, as far as mitochondrial oxidative stress homeostasis is concerned, mGSH is a vital factor maintaining mitochondrial function, controlling cell survival/death, and its depletion causes cell damage and facilitates cell death [57, 59]

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Wayner, D.D., Burton, G.W., Ingold, K.U

Since the mid-2010s, multiple studies have explored its effects on musculoskeletal structures, including skeletal muscle, connective tissue, and bone, highlighting its relevance in regenerative research models