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Recent studies using metastatic breast cancer models have demonstrated that cancer cellderived exosomal miR-138-5p can be transferred to macrophages, where it suppresses JMJD3 expression, inhibits M1 polarization, and promotes M2-like immunosuppressive phenotypes

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These findings have relevance for the role of TrxR1 acetylation in cancers with poor survival Iwasawa et al., 2011

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Chromatins were sonicated to 2001000 bp and immunoprecipitated with antibodies against NRF2, H3K4Me3, and H3K9Ac and determined with gene-specific primers (Supplementary Table 1)

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