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Expectedly, depletion of cystine or inhibition of the Xc- antiporter using synthetic small molecule inhibitors including erastin, sulfasalazine, sorafenib and even excess of glutamate triggered cystine depletion, decreased GSH level and reduced GPX4 activity, culminating in ferroptosis induction

A weak link in metabolism: the metabolic capacity for glycine biosynthesis does not satisfy the need for collagen synthesis

A convincing or probable judgement is strong enough to justify recommendations

Mitochondria-lysosome contact sites: emerging players in cellular homeostasis and disease

This structure acts as a shield and a transport vehicle

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