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The article from Prasse et al suggest[s] that chronic nicotine stimulation induced by cigarette smoking promotes macrophage and Langerhans cell accumulation in the lung via an increase in [osteopontin production]. Osteopontin and cellular receptors for nicotine may therefore be new targets for treating smoking related lung disease

While public health measures such as raising taxes on cigarettes, plain packaging and advertising controls will prevent many from taking up smoking and encourage some to quit, many of those who are most tobacco dependent will not be able to quit without treatment from a health-care professional

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