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In a high ROS environment, the diselenide bonds within the nanoclusters were compromised and decomposed into diminutive ruthenium nanoparticles, facilitating the release of nerve growth factor (NGF) and enhancing neuronal regeneration and healing of damaged neurons

Our findings reveal significant alterations in glycogen metabolism, glycolysis and lipid storage, combined with alterations in mitochondrial properties characterized by increased maximal respiratory capacity without changes in basal oxidative phosphorylation, all of which associated with the pathophysiology of PRKAG2 cardiomyopathy

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