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Xie X, Wang X, Shi X, Zhang Y, Laster KV, Liu K, et al

This reduction in GPX3 expression leads to an imbalance in extracellular redox homeostasis, ultimately promoting cancer development (46)

[15] This is exactly the biological logic that made the local MGF splice variant interesting in the first place: nature appears to use a transient, compartmentalized IGF-1 signal for repair, precisely to avoid the metabolic disturbances that broad, sustained IGF-1 receptor activation would cause including effects on glucose homeostasis

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Persistent activation of an innate immune response translates respiratory viral infection into chronic lung disease