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A1AT appears to inhibit viral infection by targeting two crucial proteases involved in the pathophysiology of the virus: the transmembrane serine protease 2 (TMPRSS2) and the ADAM17 (123, 124)

Prevention of oxidative damage and maintenance of mitochondrial function The neuroprotective behavior displayed by Sp intrigued a parallel study in the Drosophila eye since it displays detectable phenotypes in response to neurophysiological disruptions such as progressive loss of ommatidia accompanied with disrupted rhabdomere arrangement 38

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