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Interestingly, hyperglycemia has been reported to induce an increased intestinal permeability through GLUT2-dependent mechanisms and the alteration of tight junction cohesion, thus creating a leaky gut state [53]
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Animal reproduction studies with semaglutide have shown embryofetal toxicity, including structural abnormalities, at exposures below those expected at therapeutic doses in humans
However, futile quinone redox cycling can lead to a cascade of ROS formation and as such, the link between toxicity and quinone redox cycling is widely acknowledged 5
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