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Despite this mechanistic insight, the clinical assessment of zinc status remains largely confined to systemic measurements, which poorly reflect intracellular distributions or organelle-specific dysfunction

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This is where hype meets genuine cellular potential, and where clinical caution should remain

Recent studies, however, have broadened our understanding, revealing their potential roles in producing persulfides and polysulfides ( 2 S is converted to thiosulfate and sulfate (Libiad et al., 2014

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