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Induction of ferroptosis by carnosic acid-mediated inactivation of Nrf2/HO-1 potentiates cisplatin responsiveness in OSCC cells

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shorter telomeres are associated with cellular aging

Mutations of COP1 (residues K472G/H578A/Y491G) abolished its interaction with c-Jun, as demonstrated by co-transfection and reciprocal pull-down assays (Fig

Although the trial provided information about the comparison of LDLC concentrations in Graves' disease patients, no measurement of ascorbic acid levels was performed

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