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In sst 2 knockout mice, the rise in hippocampal corticosterone after an acute foot shock had a shorter onset, higher maximum and delayed recovery compared to wild type mice (Prvt et al., 4 agonist microinjected into the hippocampus in sst 2 knockout mice exposed to foot shock shortened the return to basal corticosterone levels, while not influencing the elevation to the peak rise (Prvt et al., 2 dampening the initial stress-corticosterone response, while sst 4 activation accelerates the recovery

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2025;doi:10.1001/jama.2025.21056 Wu JH, Lemaitre RN, King IB, et al

Secondary antibody, 1:3000 Alexafluor 488 goat-anti-mouse (Invitrogen: Carlsbad, Calif.) was applied for two hours at room temperature