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MOTS-c enhances memory formation and protects against neuroinflammation, with benefits observed in Alzheimers and metabolic dysfunction models [6]

The inhibition of SIRP- CD47-SHP-1 axis by using CD47 antagonist, under clinical evaluation, leads to SnCs elimination and maintain the homeostasis of tissues

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GHK-Cu (glycyl-L-histidyl-L-lysine-copper) has a unique copper-binding mechanism, where the copper ion is chelated to the tripeptide at a 1:1 molar ratio

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