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Intestinal complications of CD, such as fistula, abscesses or stenosis formation, might be also related to the decrease in the enzymatic activity of organ tissue SOD (7)

Angiostrongylus cantonensis ESPs and recombinant Calreticulin (rCRT) (a key effector protein of ESPs) induced apoptosis in human hepatoma HepG2 cells via endoplasmic reticulum stress

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Transforming growth factor-beta receptor activation represents another key mechanism underlying GHK-Cu's effects on matrix synthesis and tissue remodeling

[3] Phosphatidylcholine also plays a role in membrane-mediated cell signaling and PCTP activation of other enzymes