Instead, the selected literature was analyzed and synthesized thematically, with studies grouped according to biological pathways (e.g., monoaminergic modulation, neurotrophic signaling, HPA-axis regulation, neuroinflammation, oxidative stress, and mitochondrial function) and level of evidence (preclinical versus clinical)
doi: 10.5812/jjm.14814 317 ShuCGeLLiZChenBLiaoSLuLet al
The decreased blood and liver levels of EtOH and AA in WT in KO mice chronically fed EtOH may relate to delayed gastric emptying and/or increased gastric mucosal of EtOH-metabolizing enzymes
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