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Importantly, we further identified HO-1 mitochondrial translocation as a previously undescribed mechanism that mediates iron overload and lipid ROS accumulation within the mitochondria (Fig

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Within this network, endothelial activation and upregulation of cell adhesion molecules serve as critical downstream effectors that spatially organize leukocyte recruitment, propagate microvascular obstruction, and exacerbate tissue hypoxia

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