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Mechanistically, GHK-Cu preserved mitochondrial function by increasing mitochondrial membrane potential, alleviating age-related mitochondrial network fragmentation, shifting mitochondrial dynamics toward fusion via regulating drp-1 and fzo-1 expression, and promoting ATP biosynthesis

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[DOI] [PubMed] [Google Scholar] 45.Pavlidi N., Vontas J., Van Leeuwen T

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The role and its mechanism of intermittent fasting in tumors: friend or foe