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Existing evidence suggests that CBP and SIRT4 can promote the ubiquitin-proteasome degradation of BCAT2 by acetylation of BCAT2 at the K44 site in PDAC

In addition, VRK2 overexpression confers PC cells resistant to ferroptosis induced by exogenous ROS through promoting ER-to-Golgi trafficking of SLC7A11, which echoes the findings that PC cells with high expression of VRK2 have an elevated basal level of GSH

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Dk glutatyon seviyeleri, toksinlerin vcutta birikmesine ve kronik yorgunluk , ba ars, sindirim sorunlar gibi salk problemlerine yol aabilir

However, these studies are limited by their small sample size and failure to adjust for baseline characteristics