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Thus, the particular susceptibility of the colon to insufficient vascular perfusion[1] mandates that the main focus of the treatment of such conditions should be bypassing one or more of the vascular obstructions

clinical data is thin

A potential underlying mechanism involves attenuated phosphorylation of p53 at Ser46, which inhibits p53-mediated apoptosis and ferroptosis (Zhang et al., 2022)

A patient with MELAS syndrome combined with autoimmune abnormalities: a case report

Densitometry data were normalized with the loading control HSC70 for Cyclin D1 and CDK1, total ERK for p-ERK and JNK for p-JNK

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