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P62 in a phosphorylated form can interact with Keap1 at the same binding site for Nrf2 and thus competitively inhibits the Keap1/Nrf2 interaction resulting in Nrf2 stabilization and translocation to the nucleus

causes point mutations, and DNA breaks that cause DNA fragmentation

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The premise is that different peptides acting through different mechanisms may produce complementary or additive effects that wouldn't be observed with a single compound alone

This process is induced by the interaction between CTLA-4 expressed on Tregs or activated T cells and CD80 or CD86 molecules on the surface of antigen presenting cells (APCs) (234)

The above results showed that miRNA-324-3p could target GPX4 and be suppressed by FBI-1 in PC-3 cells