This is because the pituitary releases excessive TSH into the bloodstream in attempt to stimulate sufficient T3 and T4 production
Studies have shown that various autoantibodies, such as islet cell antibodies (ICA), glutamic acid decarboxylase antibodies (GADA), insulin antibodies (IAA), and unique self-immune responses like T-cell-mediated immune responses, are associated with the onset of T1D (Ilonen et al., 2019)
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