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Hypertension 71 , e13e115 (2018)

Translate this page into: Investigation of Microbial-mitochondrial cross-talk to explore the compatibility mechanism of Xiaojianzhong Tang against chronic atrophic gastritis rats * Corresponding author: E-mail address: [email protected] (Y

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Conditional GPX4 knockout mice exemplify the risks of single-pathway targeting: while GPX4 deletion in PT cells protects against early AKI by eliminating damaged cells, long-term deletion activates the cGASSTING pathwaylikely via release of fragmented mitochondrial DNAleading to sustained type I interferon signaling and progressive interstitial fibrosis