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G.HuangL.et al (2024)

Cytotoxicity and gene expression changes induced by inorganic and organic trivalent arsenicals in human cells

While current clinical and basic evidence strongly supports ferroptosis as a key contributing mechanism in periodontitis progression, the extent of its role relative to classical pathogenic factors such as dysbiosis and immune dysregulation, as well as its causal temporality in the natural history of the disease, remains unclear

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Figure 1: Mechanism of detection used in the GXP4 Assay Kit

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