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Although ACA exhibited no noticeable impact on early rat hepatocarcinogenesis in vivo, it suppressed the growth of HepG2 and MH1C1 cells [128]

*Correspondence: Virendra Kumar Yadav, [email protected] Dhanasekaran, [email protected] Kumar, [email protected] Disclaimer All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers

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However, metabolic malleability in invasive tumors results in recurrence as well as drug-resistant clones with more complex metabolic profiles