Modifiable risk factors in knee osteoarthritis: treatment implications. Rheumatology international 39.7 (2019): 1145-1157
To induce differentiation, cells were grown to confluence and subsequently cultured in medium as above, but without FBS and containing 0.01 mg/ml transferrin, 1 M cortisol, 200 pM triiodothyronine (T3), 20 nM human insulin, 0.25 M DEX, 500 M IBMX, and rosiglitazone (2 M until day 4, 1 M until day 8, then without rosiglitazone) (all Sigma St
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10.1039/C6MD00500D Summary Keywords NRF2, BACH1, KEAP1, oxidative stress, neurodegeneration, neuroinflammation, mitochondrial dysfunction Citation Mayer C, Riera-Ponsati L, Kauppinen S, Klitgaard H, Erler JT and Hansen SN (2024) Targeting the NRF2 pathway for disease modification in neurodegenerative diseases: mechanisms and therapeutic implications
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The practical outcome of the study by Benoist et al
