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Inflammatory signals, including IL-1, IL-6, IL-9, IL-17, interferon-gamma (IFN-), TNF-, and chemokine ligand 2 (CCL2), contribute to diminished TJs expression or misallocation [114,115,116,117,118,119]

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DOI: 10.1152/ajpheart.00982.2010 Levy MN (1971): Sympathetic-parasympathetic interactions in the heart