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Indefinite daily dosing isnt the default pulsed maintenance keeps the repair signal present without blunting the response
*Correspondence: Richard Eugene Frye, Arkansas Childrens Hospital Research Institute, Slot 512-41B, 13 Childrens Way, Little Rock, AR 72202, USA e-mail: [email protected] This article was submitted to Child and Neurodevelopmental Psychiatry, a section of the journal Frontiers in Pediatrics
Excessive ROS generation and oxidative stress exacerbate pancreatic cell injury, promote inflammatory responses, and ultimately lead to extensive pancreatic tissue damage and functional failure (117)
Acknowledgements The author is grateful to the National Institutes of Health (R35GM142882) for support which enables investigations into halogenation biochemistry in the Agarwal laboratory
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