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NADPH not only drives ATP release through coupling with OXPHOS but also maintains the GSH cycle across various metabolic pathways, thus counteracting mitochondrial lipid peroxidation and reducing susceptibility to ferroptosis (51)

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The epithelium of the non-inflamed ileum shows a normal permeability to ions and a normal paracellular permeability to 51 Cr-EDTA as compared to controls (119, 120) but is more susceptible to a challenge with sodium caprate indicating that TJs in the non-inflamed ileum of CD are more reactive to luminal stimuli contributing to the development of mucosal inflammation (120)