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Excessive ROS further mediates intestinal epithelial cell apoptosis (66) and, by inhibiting methyltransferase-like 3 (METTL3) expression, reduces the mRNA stability of pro-angiogenic factors, impeding intestinal angiogenesis (67)

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The role of CB839 in the tumor immune microenvironment has been extensively evaluated in preclinical experiments, revealing its distinct effects within the TME