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Accumulating evidence shows that activation of Nrf2 upregulates key genes involved in redox homeostasis, including SLC7A11, glutathione synthetase, and glucose-6-phosphate dehydrogenase, thereby increasing the production of reduced cofactors such as NADPH, GSH, and CoQ10, which collectively enhance cellular antioxidant capacity and counteract disease-associated oxidative stress (277)

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This synergy between transcription factors reinforces cellular redox balance, counteracts oxidative DNA damage, and inhibits melanoma cell proliferation 160

elegans model, urolithin A activates the AMPK signaling pathway and significantly promotes mitophagy, which prolongs lifespan by 45.4%