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Emerging data suggests that co-activation of both CTR and AMYRs may confer greater efficacy in metabolic control than selective activation of AMYRs 13

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DA increases renal sodium excretion, in part, by inhibiting renal NHE3, sodium phosphate cotransporters (NaPi-IIa, NaPi-IIc), Cl - /HCO 3 exchanger, sodium bicarbonate exchanger (NBCe1), NaKATPase, NCC, ENaC, and potassium channel [6]

These sleep architecture effects position DSIP as an important research compound for investigating natural sleep regulation mechanisms, developing non-addictive sleep aids, and understanding the neurochemical basis of restorative sleep physiology