Consistent with this notion, it has been reported that ER can negatively regulate the level of transferrin receptor TFRC, enhancing the resistance of ER+ breast cancer cells to ferroptosis [22]
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Others Tested These were effective, but not as consistently so as the above picks
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It is studied primarily for its ability to target mitochondria directly, stabilize the inner mitochondrial membrane, and improve cellular energy production in preclinical models of aging, cardiac injury, and metabolic disease