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As previously described in several models of inflammatory pain [21,22], our study demonstrated that cortistatin acts as an endogenous natural analgesic that is produced in response to nociceptive stimuli, including nerve injury, in parallel to nociceptive peptides to mitigate persistent painful sensitization

Dig Dis Sci 2009

J Immunol 1994

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In turn, we speculated that this impairment of mitochondrial metabolism in FoB might explain the increase in aerobic glycolysis for ATP production

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