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Meta-analysis of chitosan-mediated effects on plant defense against oxidative stress

Ketones inhibit mitochondrial production of reactive oxygen species production following glutamate excitotoxicity by increasing NADH oxidation

Specific protein 1 (SP1)-mediated upregulation of PRDX6 expression also prevented iron overload by regulating iron metabolism, restoring SLC7A11 activity and GSH activity, and further promoting the increase in GPX4 expression to prevent ferroptosis-associated DN [238]

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The product has antioxidant properties and is also effective in reducing dark spots

Consistent with the in vivo experimental results, these observations implied that Acsl4 knockout alleviated CaOX-induced mitochondrial dysfunction and reduced lipid peroxidation, thereby decreasing ferroptosis in MTECs