Its neuroprotective mechanisms encompass suppression of PI3K/Akt and TLR4/NF-B signaling, induction of heme oxygenase-1 (HO-1) expression, and regulation of anti-apoptotic/pro-apoptotic protein balance (241), suggesting it alleviates AD-associated cognitive impairment through ferroptosis inhibition (242)
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2000 During apoptosis of HL-60 and U-937 cells caspases are activated independently of dissipation of mitochondrial electrochemical potential Exp
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