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These accumulated long-chain acylcarnitines are not merely biomarkers but active pathogenic mediators

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In an immunocompetent mouse model of hepatitis B virus (HBV) infection, SFN treatment significantly promoted the repolarization of macrophages towards the antitumoral M1 phenotype, as evidenced by increased expression of Cd86 and iNOS, and inhibited the expression of Arg1

ETX with different tags (GST and 6 His) did not significantly differ in toxicities (Additional file 1: Fig

Outside the core genome, the Human-SFB-ML genomes are most similar to each other, exclusively sharing 411 gene clusters when all SFB genomes are considered (Fig