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Polymorphisms of these genes or associated regulatory sequences, or copy number variations (CNV) may contribute to a reduction in UGT activity or indeed loss of function and, therefore, reduce paracetamol glucuronidation

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Figure 7b illustrates the cardiac tissue of the positive hypercholesterolemic group (G2), which exhibits myocyte atrophy, increased fat accumulation, inflammation, fibrosis, and focal fibrotic myocitis

(B) TMRE/NAO plate reader assay depicts a significant mitochondria membrane potential depolarization induced by glutamate insult, which was dose dependently attenuated by MB