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Treatment for 3 h with RSL3-induced robust BODIPY-C11 oxidation in control cells, while Ms4a15 OE cells were unchanged (Fig

Therefore, an increase in BDNF during abstinence could activate positive neuromodulatory mechanisms as an adaptive response to excitotoxic neuronal damage

Lipid peroxidation products within these vesicles also contribute to impaired mitochondrial function in neuronal systems, highlighting a potential mechanism of toxicity following vesicle entry into the brain [141]

doi: 10.3390/cells7070084 61 BernsteinAL

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