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GPER, G-Protein-coupled estrogen receptor

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As such, zinc, which is able to function both as a signaling mediator and neurotoxin, has been implicated in cerebral ischemia

Despite these diverse origins, such as metabolic dysfunction, immune attack, toxic exposure or vascular injury, the pathophysiological mechanisms involved tend to converge on a common pathway characterized by inflammation, fibrosis, nephron loss and eventual functional failure [15,16,17,18]