The nonenzymatic initial reaction between 1 M roGFP2 WT (S 2 ) and physiological millimolar GSH concentrations was too slow to be detectable and to be of any relevance in the presence of glutaredoxins in accordance with previous analyses 32
However, disease-associated SUOX mutations have revealed that heme binding may not be strictly required for SUOX activity 100
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Elevated iron levels in the gray matter of patients with AD have been documented (1), along with dysregulated iron homeostasis and lipid peroxidation, hallmarks of ferroptosis that are implicated in AD pathology (20)
The histological improvements are consistent with reductions in oxidative stress and enhanced glycemic control, further supporting the role of FGJ in protecting key metabolic organs
