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Our in vitro results revealed that Nrf2 deactivation promoted chondrocytes senescence by upregulating p16 expression

doi: 10.3390/antiox11040745 13 AgarwalASenguptaPDurairajanayagamD

Wang J, Zhou X, Li W, Deng X, Deng Y, Niu X

Shi S, Wang L, Song C, Yao L, Xiao J

D., Partipilo, G

Notably, its role in ferroptosis is biphasic: while high, exogenously delivered concentrations of NO can induce this iron-dependent cell death, lower, endogenously regulated levels can be protective by terminating lipid peroxidation