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Mitochondria-derived methylmalonic acid aggravates ischemia-reperfusion injury by activating reactive oxygen species-dependent ferroptosis
GHK-Cu attenuated LPS-induced lung histological alterations and suppressed infiltration of inflammatory cells into lung parenchyma, demonstrating protective effects against acute inflammatory damage
Between sessions, patients continued topical minoxidil but did not use oral finasteride, dutasteride, or minoxidil during the study period
A promising target Mcl-1 belongs to a family of five proteins that play roles in controlling programmed cell death, or apoptosis
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