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These ailments stand as the primary cause of elevated morbidity and mortality on a global scale [1]

EIF2, the eIF2 subunit of eIF2, was subsequently activated, leading to the activation of the GCN2-ATF4 signaling pathway [228]

Using targeted proteomics, ENPP-2 was increased in cerebrospinal fluid (CSF) of AD patients with amnestic mild cognitive impairment (aMCI), and ENPP-2 directly reflects the brain glucose steady state (42, 43)

CO23MCFG showed a behaviour similar to that of the sensitive strain

Rhee, A.M